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University Warsawa

University Warsawa

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22 Projects, page 1 of 5
  • Funder: NWO Project Code: 462-14-153

    This project aims to understand the role of welfare systems in destination and origin countries for migration patterns within and towards Europe. Welfare states were developed and associated with the nation state, explaining why provisions remain predominantly linked to nationality and residency. The project moves beyond prior studies on the contested existence of welfare magnets and the presumed threat of (low-skilled) migration to the viability of welfare state benefits. A receiving country bias has caused research to neglect the important role of welfare regimes in origin countries on migration aspirations and decisions. Furthermore, little empirical knowledge of the effects of transferability of welfare entitlements on mobility in Europe exists. To fill these gaps and understand how growing levels of mobility intersect with existing welfare regimes across Europe, the project addresses three research questions: How and to what extent do welfare systems affect mobility patterns in Europe? To what extent and how do perceptions of access to welfare arrangements in origin and destination countries shape migration decisions? What role does transferability of welfare accounts play in mobility across Europe? The project combines macro and micro perspectives, and applies a mixed-methods approach of innovative analysis of existing statistics and migration data added with new primary data collection via case studies in seven countries. It goes beyond reductionist categorisations of receiving and sending countries by considering all case study countries simultaneously as origins and destinations.

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  • Funder: WT Project Code: 067504
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  • Funder: FCT Project Code: SFRH/BD/65372/2009
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  • Funder: FCT Project Code: SFRH/BSAB/1286/2012
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  • Funder: WT Project Code: 084885
    Funder Contribution: 277,749 GBP

    In early development, during the period from the meiotic maturation of oocytes to eggs through to onset of zygotic transcription in the fertilised embryo, the principal means of gene regulation is translational control of the masked maternal mRNA. The cytoplasmic polyadenylation element binding protein (CPEB) is a critical regulator of translation in early development, in organisms ranging from clam to man. CPEB binds CPE elements in the 3' untranslated regions of specific mRNAs, and controls t heir expression at the level of translational repression in oocytes, and by cytoplasmic polyadenylation and translational activation in eggs. In Xenopus oocytes, CPEB is present in a large RNP complex with several highly conserved RNA-binding partners, including Xp54 RNA helicase, Pat1, and RAP55, and inhibits cap-dependent protein synthesis using a novel pairing of the eIF4E-binding protein 4E-T(ransporter) and eIF4E1b, a close homologue of the canonical cap-binding protein. Our aims are to: I. Investigate eIF4E1b interactions with the m7GpppG cap II. Investigate eIF4E1b interactions with 4E-T and eIF4G III Identify mRNAs regulated by eIF4E1b IV. Assess the function of eIF4E1b in translational control V. Investigate how the interaction between eIF4E1b and 4E-T change during meiotic maturation when translation is activated.

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